Chest
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

Guest Access | Sign In via User Name/Password
This Article
Right arrow Full Text (PDF) Free
Right arrow Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when eLetters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Article Archive
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Williams, P.
Right arrow Articles by Smyth, L.
Right arrow Search for Related Content
PubMed
Right arrow Articles by Williams, P.
Right arrow Articles by Smyth, L.

Chest, Vol 76, 566-570, Copyright © 1979 by American College of Chest Physicians

ARTICLES

Symptomatic coccidioidomycosis following a severe natural dust storm. An outbreak at the Naval Air Station, Lemoore, Calif

PL Williams, DL Sable, P Mendez and LT Smyth

Eighteen newly diagnosed cases of symptomatic cocidioidomycosis developed two to four weeks following exposure to a severe natural dust storm. The population at risk consisted of 26,000 residents of the San Joaquin Valley with access to health care at the Naval Hospital, Lemoore, Calif. Eight patients were white, and ten were nonwhite. The number of cases per 100,000 was estimated to be 36 for the white group and 254 for the nonwhite group. The disease was disseminated in four patients, and all were from the nonwhite group. One patient with disseminated disease, a black man, died. These data suggest that nonwhites may be relatively more susceptible to acquiring primary disease, in addition to developing disseminated disease. Dust storms of this magnitude must be considered a threat to health for populations living within areas endemic for coccidioidomycosis.


This article has been cited by other articles:


Home page
 Infect. Immun.Home page
M. d. P. Jimenez, L. Walls, and J. Fierer
High Levels of Interleukin-10 Impair Resistance to Pulmonary Coccidioidomycosis in Mice in Part through Control of Nitric Oxide Synthase 2 Expression.
Infect. Immun., June 1, 2006; 74(6): 3387 - 3395.
[Abstract] [Full Text] [PDF]

Home page
 Clin. Microbiol. Rev.Home page
R. A. Cox and D. M. Magee
Coccidioidomycosis: Host Response and Vaccine Development
Clin. Microbiol. Rev., October 1, 2004; 17(4): 804 - 839.
[Abstract] [Full Text] [PDF]

Home page
 J. Clin. Microbiol.Home page
N. F. Crum, M. Potter, and D. Pappagianis
Seroincidence of Coccidioidomycosis during Military Desert Training Exercises
J. Clin. Microbiol., October 1, 2004; 42(10): 4552 - 4555.
[Abstract] [Full Text] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1979 by the American College of Chest Physicians.