Chest
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

Guest Access | Sign In via User Name/Password
This Article
Right arrow Full Text (PDF) Free
Right arrow Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when eLetters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Article Archive
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Mallery, J.
Right arrow Articles by Henry, W.
Right arrow Search for Related Content
PubMed
Right arrow Articles by Mallery, J.
Right arrow Articles by Henry, W.

Chest, Vol 100, 470-473, Copyright © 1991 by American College of Chest Physicians

ARTICLES

Effects of heart rate and pulmonary artery pressure on Doppler pulmonary artery acceleration time in experimental acute pulmonary hypertension

JA Mallery, JM Gardin, SW King, S Ey and WL Henry
Division of Cardiology, University of California Irvine, Orange 92668.

Chronic pulmonary hypertension in humans is characterized by shortening of the pulmonary artery acceleration time as measured by Doppler echocardiography, such that the higher the pulmonary artery pressure, the shorter the pulmonary acceleration time. Increases in heart rate are also known to produce decreases in the pulmonary artery acceleration time. To explore the relationship between mean pulmonary artery pressure, heart rate, and Doppler pulmonary artery acceleration time, experimental acute pulmonary hypertension was created in nine Duroc swine, either by infusion of Sephadex beads with embolization of the pulmonary arterial circulation or by partially occluding the main pulmonary artery 8 to 10 cm distal to the pulmonic valve. Pulmonary artery Doppler flow velocity recordings and invasive pressure measurements were made at baseline and at paced atrial rates ranging from 60 to 160 beats per minute, in 20-beat increments. The results in this acute animal model reveal that increases in heart rate produced significant decreases in Doppler pulmonary artery acceleration time at mean pressures below 25 mm Hg. However, with mean pulmonary artery pressures greater than 25 mm Hg, both heart rate and increases in pulmonary artery pressure had no significant effect on acceleration time.


This article has been cited by other articles:


Home page
 RadiologyHome page
E. Mousseaux, J. P. Tasu, O. Jolivet, G. Simonneau, J. Bittoun, and J.-C. Gaux
Pulmonary Arterial Resistance: Noninvasive Measurement with Indexes of Pulmonary Flow Estimated at Velocity-encoded MR Imaging-Preliminary Experience
Radiology, September 1, 1999; 212(3): 896 - 902.
[Abstract] [Full Text]

Home page
 Journal of Diagnostic Medical SonographyHome page
S. K. Miller
Pulsed Doppler Echoca rd iographic Measurement of Pulmonary Acceleration Time in Diagnosing Patients With Pulmonary Hypertension
Journal of Diagnostic Medical Sonography, September 1, 1995; 11(5): 241 - 245.
[Abstract] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1991 by the American College of Chest Physicians.